EMF Blocking plus EMF Shielding

· 2 min read
EMF Blocking plus EMF Shielding

If you reside in an apartment or house or just want to keep your home free of electromagnetic fields, there are a number of ways you can reduce exposure. One of the most effective is to reduce your electronic device use. You can also turn to EMF block paint to stop EMF radiation from reaching your home.  emf blocker  to shield your home from EMF radiation would be to put up a RF shielding canopy. It is a type made of net that has EMF shielding. It's used to block EMFs from entering a room. Another alternative is to have your home equipped with an enclosure that is conductive. They are referred to as Faraday cages.

Numerous studies have demonstrated how the non-ionizing energy of RF has antiproliferative properties in HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is believed be based on the downregulation of cancer stem cells. This may account for the long-term response seen in some patients with advanced HCC. But, the reason for AM EMF's impact on patients suffering from cancer isn't evident.


Aspects of AM RF EMF on HCC tumour growth in vivo was studied in mice. The tumors were split into 3 groups.  emf blockers  was not exposed RF EMF. Second group members were exposed to RF EMF at the same frequency to that used in humans. In the third, they were subjected to RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on the tumours was assessed against the effect of RCF. The results revealed that cancers treated with HCCMF were significantly shrinking. However, the tumors treated with RCF did not show any evidence of shrinkage in the tumour.

The mechanism of tumor-specific AM RF EMF may be due to the fact that cancer cells require Cav3*2 T-type voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effect on HCC cells is caused by CACNA1H the protein which regulates the Ca2+ influx specific to tumors. The results suggest that CACNA1H could have wider implications in the treatment and diagnosis of many cancers.

The tumors in the control group were not exposed RF EMF, and were fed a normal diet of mice. The tumours in the HCCMF group were treated with Huh7 cells after they were five-seven weeks old. The tumors were removed after they had a high burden.

The tumours from the three groups showed distinct growth curves. The tumours treated with HCCMF had a significant reduction in size of the tumor after 8 weeks. However, tumors treated with RCF showed no shrinkage. The difference was highly significant. The tumors treated with RCF showed necrosis, which is typical when tumors are that are exposed to RCF. It is possible that this necrosis was caused by an absence of oxygen in the larger cancers.

In summary, the results show the fact that AM EMF has anticancer activity in vitro and in live.  block emf  of studies have proven the fact that AM RF EMF produces measurable shrinkage of tumors for HCC patients. It is possible that AM RF EMF causes these effects because of CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. Furthermore, AM RF EMF may exert a sustained influence on the growth of HCC tumours in vivo.