No matter if you live in a house or apartment or simply would like to ensure your home is free of EMFs There are a variety of methods to reduce exposure. One of the most effective is to restrict the use of electronic devices. You can also turn to EMF block paint to prevent EMF radiation from entering your home. Another way to shield your house from EMF radiation would be to use a RF shielding canopy. It is a type of net that contains EMF shielding and is used to stop EMFs from entering a space. Another alternative is to have your home fitted with an enclosure that is conductive. These enclosures are known as Faraday cages.
A number of studies have proven how the non-ionizing energy of RF has antiproliferative effects on HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is thought to result from the deregulation of cancer stem cells. This may account for the long-term responses seen in patients suffering from advanced HCC. However, the mechanism behind AM EMF's impact on cancer patients is not yet clear.
Effects of AM electromagnetic fields (RFEM) on HCC tumour growth in vivo were studied in mice. The tumours were divided into 3 groups. First, the group that was unaffected to RF EMF. block emf were exposed RF EMF at a frequency that is similar to that used in humans. Third group members were exposed RF EMF at HCC-specific modulation frequencies. The effects of HCCMF on the tumours was compared to that of RCF. emf blocker indicated that cancers treated with HCCMF showed significant shrinkage. However, the tumours treated with RCF did not show any evidence of shrinkage of the tumor.
The reason for cancer-specific AM RF EMF might be due to the fact that cancer cells require Cav3*2 T-type voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effect upon HCC cells is caused through CACNA1H which is a protein that regulates the Ca2+ influx specific to tumors. The results suggest that CACNA1H may have broader implications for the treatment and diagnosis of many cancers.
The tumors in the control group were not exposed to EMF from radiofrequency, and fed a standard mouse diet. The tumors of those in the HCCMF group were injected with Huh7 cells at the time they were five-seven weeks old. emf blocking were then killed when they showed excessive burden.
The tumours from the three groups also displayed different growth curves. The tumors treated with HCCMF had a significant reduction in size of the tumor after 8 weeks. However, tumors treated with RCF showed no shrinkage. The difference was highly significant. The tumors treated by RCF showed necrosis that is common in tumors that have been exposed to RCF. The possibility is that this necrosis was due to an absence of oxygen in the more invasive tumors.
In sum, the results indicate an AM-RF EMF has anticancer properties in vitro as well as in the vivo. A number of studies have proven it is true that AM RF EMF produces measurable shrinkage of tumors in HCC patients. It is possible that AM RF EMF causes these effects due to CACNA1H which is a protein involved in the process of tissue-specific Ca2+ influx. Furthermore, AM RF EMF may cause a lasting impact on the growth of HCC tumours in living tissue.