If you reside in an apartment or a house, or you just would like to keep your home free from EMFs There are plenty of methods to reduce exposure. One of the easiest is to reduce the use of electronic devices. block emf is also possible to use EMF block paint to stop EMF radiation from entering your house. Another easy way to protect your home from EMF radiation would be to install a RF shielding canopy. It's a kind of net that contains EMF shielding and is used to block EMFs from entering a space. Another option is to have your home fitted with an enclosure that is conductive. These devices are called Faraday cages.
A number of studies have proven that the non-ionizing RF EMF has anti-proliferative effects on HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed involve down-regulation in cancer-related stem cells. This could be the reason for the long-term response seen in some patients with advanced HCC. But, the reason for AM EMF's impact on cancer patients is not yet clear.
Effects from AM electromagnetic fields (RFEM) on HCC tumour growth in vivo were studied in mice. The tumors were divided in three different groups. First, the group that was unaffected to RF EMF. The second group was exposed to RF EMF at the same frequency to that used in humans. block emf were exposed to RF EMF with HCC-specific modulation frequencies. The effect of HCCMF on the tumours was assessed against the effect of RCF. The results showed that the tumours treated with HCCMF were significantly shrinking. However, tumours treated with RCF didn't show evidence of shrinkage in the tumour.
The mechanism of tumor-specific AM RF EMF might be driven by the fact that tumour cells require Cav3*2 T-type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation on HCC cells is caused by CACNA1H the protein which mediates tumour-specific Ca2+ influx. The results suggest that CACNA1H could have wider implications in the treatment and diagnosis of various cancers.
The tumors in those in the group that were unaffected to EMF from RF, and fed a normal mouse diet. The tumors in the HCCMF group were treated with Huh7 cells when they were between five and seven weeks old. The tumors were removed when they showed excessive burden.
The tumors in the three groups showed distinct growth curves. The HCCMF-treated tumors showed a significant decrease in the size of the tumour after 8 weeks. However, the tumours treated with RCF didn't show signs of shrinkage. The difference was highly significant. emf blockers treated with RCF were able to show necrosis that is common in tumors that have been that are exposed to RCF. There is a possibility that this necrosis was due to the lack of oxygen in larger tumours.
In summary, the results suggest an AM-RF EMF is a powerful source of anti-cancer properties in vitro as well as in vivo. Numerous studies have demonstrated it is true that AM RF EMF produces measurable reduction in tumours in HCC patients. The possibility is that the AM EMF produces these effects because of CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. Furthermore, AM RF EMF may have a long-lasting effect on the development of HCC tumors in vivo.