If you reside in an apartment or house or just would like to keep your home free from EMFs, there are a number options to limit your exposure. One of the most effective is to restrict the use of electronic devices. You can also turn to EMF block paint to prevent EMF radiation from entering your home. Another method to protect your home against EMF radiations is to put up an RF shielding cover. It is a type made of net which contains EMF shielding and is used to prevent EMFs from entering a space. Another option is to have your home equipped with an electrical enclosure. These devices are called Faraday cages.
A number of studies have proven that the non-ionizing RF EMF produces anti-proliferative effects on HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is thought to involve down-regulation in cancer-related stem cells. This may account for the long-term response seen in certain patients suffering from advanced HCC. However, the reason behind AM EMF's effects on patients with cancer is not clear.
Aspects on the effects of AM RF EMF on HCC tumor growth in vivo were examined in mice. The tumours were divided into 3 groups. First, the group that was unaffected to RF EMF. Another group of participants was subjected to RF EMF at the same frequency to that of humans. Third group members were exposed RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on tumors was compared to that of RCF. The results indicated that tumors treated by HCCMF showed significant shrinkage. However, the tumours treated with RCF showed no evidence of shrinkage of the tumor.
The mechanism of tumour-specific AM RF EMF might be based on the fact that tumor cells require Cav3*2 type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation in HCC cells is mediated by CACNA1H the protein which regulates the Ca2+ influx specific to tumors. The results suggest that CACNA1H may have broader implications for the diagnosis and treatment of a variety of cancers.
The tumors in the controls were never exposed EMF from RF, and fed a normal mouse diet. https://eggnogflight26.doodlekit.com/blog/entry/25139493/emf-blocking-and-even-emf-shielding in HCCMF HCCMF group were injected with Huh7 cells at the time they were 5 to 7 weeks old. emf blockers were removed in cases of excessive burden.
The tumours in the three groups also displayed different growth curves. The tumors treated with HCCMF saw a significant decrease in size of the tumor after 8 weeks. However, the tumours which were treated by RCF didn't show shrinkage. The difference was significant. The tumors treated by RCF had necrosis, which is typical in tumors that have been that are exposed to RCF. There is a possibility that the necrosis is caused by the lack of oxygen in the larger cancers.
In summary, the results suggest an AM-RF EMF has anticancer activity in vitro and in the vivo. Several studies have shown the fact that AM RF EMF produces measurable shrinkage of tumors within HCC patients. It is possible that AM RF EMF causes these effects because of CACNA1H which is a protein involved in the tissue-specific Ca2+ influx. In addition, AM RF EMF may cause a lasting impact on the development of HCC tumours in the vivo.